High-flow nasal oxygen (HFNO) is the administration of heated, humidified and blended
air/oxygen via nasal cannula at rates ≥ 2 L/kg/min. HFNO is an open system that can be
used with nasal prongs of different sizes and was developed in neonatal intensive care
unit for preterm babies with apnoea as alternative to continuous positive airway pressure
(CPAP). Due to its ease of use and safety to apply to a wide range of indication HFNO is
increasingly gaining interest for providing respiratory support in paediatric patients
and in adults in ICU with respiratory failure. In adult populations, the use of HFNO
permits to prevent desaturation during tracheal intubation of intensive care patients
with mild-to-moderate hypoxemia. An application for HFNO in adults and children, is the
extension of safe apnoea in patients who were undergoing general anaesthesia for
hypopharyngeal or laryngo-tracheal surgery. This method, the so-called safe apnoeic
oxygenation, also prevents hypoxemia in children during intubation. By using this
technique, Patel et al. demonstrated a significate prolongation of apnoea time and
proposed a ventilatory effect, as these studies revealed a slower increase in pCO2 than
physiologically was expected. In these studies, researchers compared their data to
studies from the 1950-ies, where CO2 increase during apnoea was investigated. In
contrast, the investigators' previous research projects with HFNO did not confirm the
claimed ventilatory effect in children and adults.
Furthermore studies performed in spontaneously breathing neonates and adults have shown
the ability of HFNO to generate some increase in pharyngeal pressure, which could explain
the improvement of oxygenation despite prolongation of apnea time. The investigators'
previous study on adult patients showed that a relevant increase of pressure was nearly
absent while patient's mouth was open. Currently, there is no data on the physiological
pressure that is generated in the subglottic airway in apneic children treated with HFNO.
The traditional measurement of intratracheal pressure with a catheter in the trachea is
considered to pose a risk in small children.
The main objective of this study is thus to investigate the variations of poorly
ventilated lung units (i.e., silent spaces) as a surrogate for functional residual
capacity measured by electrical impedance tomography to dynamically assess atelectasis
formation and regression under apnoeic oxygenation with different flow rates.
Eligible children will receive premedication with Midazolam rectal/oral 0.5 mg/kg or
Dexmedetomidine nasal 2 mcg/kg 30 minutes before the beginning of the procedure (local
SOPs of the paediatric anaesthesia departments). Mandatory monitoring will consist of
non-invasive peripheral oxygen saturation (SpO2), heartrate (HR), and non-invasive blood
pressure (NIBP). An intravenous line for drugs injection will be placed.
After start of anaesthesia (="induction"), adequate face-mask ventilation will be
established. The sealed envelope for randomisation will then be opened. Standard
anaesthesia will be continued using of intravenous propofol. Anaesthetic depth will be
assessed using NarcotrendTM (NarcotrendTM, Hannover, Germany), maintaining values between
40 and 60. Additional study related non-invasive monitoring: transcutaneous tcCO2 and O2
(ToscaTM, Radiometer, Neuilly-Plaisance, France) measurement, thoracic electrical
impedance tomography (EIT, PulmoVista 500, Draeger, Luebeck, Germany) and NIRS
(Niro-200NX (Hamamatsu, Tokyo, Japan). ECG, pulse-oximetry, blood pressure, Narcotrend
(NarcotrendTM, Hannover, Germany), thoracic EIT will be measured continuously, starting
before induction while spontaneous breathing and ending 1 minute after the
recruitment-manoeuvre. All patients will receive neuromuscular blockade medication of 2 x
ED95 (standard intubation dose) to facilitate airway management. Neuromuscular block will
be assessed using train-of-four (TOF) monitoring (TOF-Watch, Organon Ltd, Dublin,
Ireland). A TOF value of zero before apnoea start and throughout the whole procedure will
be deemed essential.
After that one minute of pressure support mask ventilation (Pmax 20 cm H20) with a backup
respiratory rate of 20/min, normalized at a volume of 6-8 ml.kg-1 with 100% oxygen and
will be applied. The ventilation will be discontinued, and the child will be left apnoeic
for 5 minutes receiving oxygen according to the randomisation.
Children will be randomized to receive three different flow rates of 100% oxygen, warmed
and humidified with the OptiFlow device (Fisher&PaykelTM, Auckland, New Zealand):
group 1): 0.2 l/kg/min + continuous jaw thrust
group 2): 2 l/kg/min + continuous jaw thrust
group 3): 4 l/kg/min + continuous jaw thrust (control group)
Group 4): 2 l/kg/min with OptiFlow FiO2 1.0 using OptiFlow-Switch system by
Fisher&Paykel.
The nostrils must not be occluded by the nasal cannula by more than 50%. The time until
desaturation from SpO2 100% to SpO2 95% will be measured. A chest ultrasound at end of
intervention after definitive airway management will prove that no pneumothorax developed
during the procedure.
Break-up criteria during apnoea are: SpO2 below 95%, transcutaneous CO2 above 70 mmHg, or
time of apnoea >5 minutes, a decrease of NIRS >30% from baseline.